The caseous necrosis is the basic process of tuberculosis disease in humans. The interval from infection to tuberculin conversion is never more than 8 weeks and in general is 5 to 7 weeks (21). The onset of caseous necrosis coincides with the development of acquired immune resistance or CMI and DTH.
How does Caseous necrosis occur?
Causes. Frequently, caseous necrosis is encountered in the foci of tuberculosis infections. It can also be caused by syphilis and certain fungi. A similar appearance can be associated with histoplasmosis, cryptococcosis, and coccidioidomycosis.
When does necrosis occur?
It occurs when too little blood flows to the tissue. This can be from injury, radiation, or chemicals. Necrosis cannot be reversed. When large areas of tissue die due to a lack of blood supply, the condition is called gangrene.
When does coagulative necrosis occur?
Coagulative necrosis generally occurs due to an infarct (lack of blood flow from an obstruction causing ischaemia) and can occur in all the cells of the body except the brain. The heart, kidney, adrenal glands or spleen are good examples of coagulative necrosis.
What causes Colliquative necrosis?
Liquefactive necrosis (or colliquative necrosis) is a type of necrosis which results in a transformation of the tissue into a liquid viscous mass. Often it is associated with focal bacterial or fungal infections, and can also manifest as one of the symptoms of an internal chemical burn.
Where is Caseous necrosis found in the body?
Caseous necrosis is more frequently found in the mesenteric nodes than in intestinal tissue itself.